Logo image
Selective localization of collybistin at a subset of inhibitory synapses in brain circuits
Journal article   Open access   Peer reviewed

Selective localization of collybistin at a subset of inhibitory synapses in brain circuits

A Patrizi, L Viltono, E Frola, K Harvey, Robert J Harvey and M Sassoè-Pognetto
Journal of Comparative Neurology, Vol.520(1), pp.130-141
2012
pdf
PDF - Author's Accepted Version409.06 kBDownloadView
Accepted VersionPDF - Author Accepted Version Open Access
url
https://doi.org/10.1002/cne.22702View
Published Version

Abstract

gephyrin GABAAreceptor receptor clustering GABAergic synapses
Collybistin is a brain-specific guanine nucleotide exchange factor (GEF) that is crucial for the postsynaptic accumulation of gephyrin and γ-aminobutyric acid A receptors (GABAARs) at a specific subset of inhibitory synapses. Our understanding of the in vivo function of collybistin has been hampered by lack of information about the synaptic localization of this protein in brain circuits. Here we describe the subcellular localization of endogenous collybistin by using antibodies raised against distinct molecular domains that should recognize the majority of endogenous collybistin isoforms. We show that collybistin co-clusters with gephyrin and GABAARs in synaptic puncta and is recruited to postsynaptic specializations early during synapse development. Notably, collybistin is present in only a subset of gephyrin-positive synapses, with variable co-localization values in different brain regions. Moreover, collybistin co-localizes with GABAARs containing the α1, α2, or α3 subunits, arguing against a selective association with specific GABAAR subtypes. Surprisingly, we found that collybistin is expressed only transiently in Purkinje cells, suggesting that in these cerebellar neurons collybistin plays a selective role during the initial assembly of postsynaptic specializations. These data reveal a remarkable heterogeneity in the organization of GABAergic synapses and provide an anatomical basis for interpreting the variable effects caused by disruption of the collybistin gene in human X-linked intellectual disability and mouse knockout models. © 2011 Wiley Periodicals, Inc.

Details

Metrics

32 File views/ downloads
225 Record Views

InCites Highlights

These are selected metrics from InCites Benchmarking & Analytics tool, related to this output

Collaboration types
International collaboration
Web Of Science research areas
Neurosciences
Zoology

UN Sustainable Development Goals (SDGs)

This output has contributed to the advancement of the following goals:

#3 Good Health and Well-Being

Source: InCites

Logo image