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Hemolytic disease of the fetus and newborn caused by anti-sD antibody in a GP.Mur/Mur Thai mother and review of the prevalence of sD in Thai blood donors
Journal article   Open access   Peer reviewed

Hemolytic disease of the fetus and newborn caused by anti-sD antibody in a GP.Mur/Mur Thai mother and review of the prevalence of sD in Thai blood donors

Genghis H Lopez, Morakot Emthip, Ploymanee Suwanwootichai, Glenda M Millard, Brett Wilson, Sunisa Onpuns, Kanchana Laemsri, Pimol Chiewsilp, Robert Lewis Powell Flower, Catherine A Hyland, …
Transfusion, Vol.62(10), pp.2137-2142
2022
PMCID: PMC9826107
PMID: 36062546
Appears in  UniSC Diversity and Inclusion Research Collection
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Hemolytic disease of the fetus and newborn caused by anti-sD antibody in a GP.Mur-Mur Thai mother and review of the prevalence of sD in Thai blood donors892.54 kBDownloadView
Published Version Open Access CC BY-NC-ND V4.0
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https://doi.org/10.1111/trf.17086View
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Abstract

MNS blood group system anti-sD haemolytic disease of the fetus and newborn prevalence of sD in Thai blood donors sD antigen UniSC Diversity Area - Life Stages
Background: Low-prevalence antigen sD (MNS23) is encoded by GYPB c.173C > G. Hemolytic disease of the fetus and newborn (HDFN) due to anti-sD is rare. A mother delivered a newborn whose red blood cells (RBCs) were DAT-positive and was later diagnosed with HDFN. Serum from the mother was incompatible with the father's RBCs and was used to screen 184 Thai blood donors. This study aimed to investigate the cause of HDFN in a Thai family and determine the prevalence of sD in Thai blood donors. Materials and methods: Three family members and four blood donors were investigated in the study. Massively Parallel Sequencing (MPS) was used for genotyping. Standard hemagglutination techniques were used in titration studies, phenotyping, and enzyme/chemical studies. Anti-s, anti-Mia , anti-JENU, and anti-sD reagents were used in serological investigations. Results: The mother was GYP*Mur/Mur. The father and the four donors were GYPB*s/sD predicting S - s + sD +. The baby was GYP*Mur/sD and his RBCs were Mia +, s + w with anti-s (P3BER) and JENU+w . RBCs from two GYPB*sD -positive blood donors reacted with anti-sD (Dreyer). Proteolytic enzyme α-chymotrypsin-treated sD + cells did not react with anti-sD (Wat) produced by the GP.Mur/Mur mother but reacted with the original anti-sD (Dreyer). Discussion: This is the first report of HDFN due to anti-sD in the Asian population. The genotype frequency for GYPB*sD in a selected Thai blood donor population is 2.2% (4/184). Anti-sD should be considered in mothers with Southeast Asian or East Asian background when antibody identification is unresolved in pregnancies affected by HDFN.

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