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Investigation of the protective effects of omega-3 fatty acids in a mouse model of abdominal aortic aneurysm
Conference presentation

Investigation of the protective effects of omega-3 fatty acids in a mouse model of abdominal aortic aneurysm

Fraser D Russell
USC Research Conference, 2013 (Sunshine Coast, Australia, 01-Jul-2013–05-Jul-2013)
University of the Sunshine Coast
2013
url
https://www.usc.edu.au/View
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Abstract

Clinical Sciences abdominal aortic aneurysm omega-3 fatty acids
Abdominal aortic aneurysm (AAA) is an inflammatory cardiovascular disease that is characterised by destruction of the aortic wall. Mortality rates are high in patients experiencing dissected or ruptured abdominal aorta. Surgical repair reduces mortality, but is limited by a need for regular follow-up and post-surgical complications (Holt et al., 2012). Pharmacological therapy is limited by poor efficacy (Golledge & Norman, 2011), and so new strategies to manage patients with AAA are required. Omega-3 fatty acids (n-3 FAs), and their metabolites (e.g. resolvin D1), have anti-inflammatory and/or pro-resolving effects. In contrast, n-6 FAs have pro-inflammatory properties. We hypothesise that ahigh dietary intake of n-3:n-6 FAs may attenuate the progression of inflammatory disease and reduce mortality in AAA. We have investigated this using apolipoprotein E-knockout (ApoE-/-) mice fed a high (HD) or low ratio n-3:n-6 FA diet (LD) for 8 weeks prior to infusion with angiotensin II (1000 ng/kg/min) for 2-14 days (to induce AAA). The n-3:n-6 FA ratio in erythrocyte membranes of HD mice (0.577±0.020 : 1; n=8) was higher than for LD mice (0.105±06 : 1; n=5, P<01). The n-3 index for HD mice (12.94±0.44%, n=8) was higher than for LD mice (2.88±0.17%, n=5, P<01), confirming incorporation of the FAs into membrane phospholipids. HD mice also had a higher plasma concentration of resolvin D1 (743±76 pg/ml, n=9) compared to LD mice (457±60 pg/ml, n=9, P<0.01). Mortality from aortic rupture was 4/20 in HD mice (2 abdominal, 2 thoracic ruptures) and 7/20 in LD mice (7 abdominal, 0 thoracic ruptures). Our study also compares HD and LD mice for structural changes in the aortic wall (e.g. elastin fragmentation, superoxide and peroxynitrite formation). Our findings suggest protection against AAA rupture by dietary supplementation of ApoE-/- mice with feed containing a high ratio of n-3:n-6 FAs.

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